In Alzheimer’s disease, glutamate excitotoxicity which can damage neurons through chronic activation is exacerbated by deficiency of ABCA7, the second strongest genetic risk factor for the disease. The findings highlight ABCA7 and glutamate–GABA signaling as potential therapeutic targets.

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和黄医药这款首创药栽在安全性上

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}Note: function parameters and return types always require type annotations. There is no inference for function signatures:

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